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by Milos Pokimica ND

by Milos Pokimica ND

Learn About Nutrition

Cooked meat bacteria endotoxemia- Inflammation and diet

"Enlarged inflammation from meat but not from plant food is a consequence of the toxic load of dead meat bacteria endotoxins. Dead bacteria also count as toxins."

We as humans, unlike carnivores species, have very low resistance filter meaning any live bacteria we eat will be creating inflammation and diet can kill us if bacteria is dangerous. We cannot eat uncooked meat. Carnivorous animals have extremely corrosive bile acids that are able to kill any living microorganism and their digestive system is practically sterile. We as any other plant eater have mild bile acid and low resistance filter and have to cook meat and any other food item that has the potential to spread an infectious disease. For example, pasteurization is mandatory.

What people don’t understand is that even if we do cook meat we do not magically dematerialize all of the bacteria that were present in there. We only kill them by heat but they are still in there. Microorganisms are still in the meat just dead. They will not pose any risk of infection anymore.

But that does not mean that they don’t pose a risk anymore.

Even dead non-probiotic bacteria do count as toxins.

Some of the world’s most toxic substances are these dead meat bacteria endotoxins.

what is endotoxin
Endotoxin effects

These substances known as endotoxins (Greek éndon within; cognate with Old Irish ind-) are thermally (250C) and chemically stable and extremely toxic. Endotoxin is a complex lipopolysaccharide (LPS) found in the outer cell membrane of gram-negative bacteria (E.coli, Salmonella typhi, Shigella).

Bacteria shed endotoxins in large amounts upon cell death. Meaning, the bacteria can be dead or cooked for a long time, but their endotoxins are still there. Endotoxins are chemically very stable and can withstand our body’s best attempts at acid and enzyme degradation. One of the leading causes of hundreds of studies that display enlarged inflammation from animal foods, but not from most plant foods, maybe is a consequence of a toxic load of dead bacteria endotoxins in animal products. These bacteria shed endotoxins after death and then when we eat them they are absorbed into our system, leading to the endotoxemia inflammation we see after egg, meat, and dairy consumption, as well.

Endotoxin representation
Endotoxin representation

This would cause damage to our internal organs and the entire body and will increase the chances of chronic diseases (1).

If we already have an autoimmune disease such as atherosclerosis, for example, this will just agitate our immune system even worse and would create even more of an immune response (2).

Here is one study that discovered a link between endotoxin exposure and diabetes type 2 (3).

Endotoxin effects
Endotoxin effects

What a low level of chronic endotoxemia inflammation does is that it causes damage like any other inflammation just in a prolonged period. What that translates to is faster and more noticeable DNA damage, a higher mortality rate from chronic diseases, and decreased longevity.

In contrast, plant foods do not show this trait, and actual consumption is correlated with the anti-inflammatory reaction after a meal because of the antioxidants and other anti-inflammatory phytochemicals. It would be interesting to see how much inflammation meat consumption causes in carnivorous species. So far I was unable to find research that looks into dead meat bacteria endotoxemia exposure in carnivorous species. This could be potentially interesting because if meat causes no inflammation in carnivorous animals, we might look at a way how to lower the same inflammation in our own body. 

Consumption of meat, therefore, is associated with an increase in inflammation even if we disregard the risk of live infectious bacteria. This mechanism is natural and normal, and all of the carnivorous species had it to some extent but are more adept at coping with it.

Types-of-inflammation
Types of inflammation

A fresh hamburger contains approximately a hundred million bacteria per quarter pounder. Eating meals high in bacterial endotoxins could develop mild but systemic inflammatory episodes that predispose subjects to the development of chronic diseases.

The animal fat that comes in the same package may play a role in the pathogenesis of this after-meal inflammation. Endotoxins hold a powerful attraction for the saturated fat, so they stick to it and then get absorbed through the gut wall and into the bloodstream (4).

Would this happen if we eat food that is high in saturated fat from plant origin? It would, but the difference is that there are no high levels of these toxins in plant-based foods. For example, cocoa has a high-fat content. It is one of the plants that has energy stored in a form of saturated fat, the same fat that is found in the animal kingdom. But cocoa also has a large number of antioxidants and in studies always decreases the level of C-reactive protein in subjects (a marker for inflammation) (5)(6).

The high antioxidant content of cocoa prevails and is able to neutralize the pro-inflammatory effects of endotoxins that are not present in the plants in high numbers, to begin with (7).

The problem with meat is a high concentration of bacteria. This means that eating a standard Western diet rich in animal protein and refined sugar and fat will require a much higher level of antioxidant to negate the bed pro-inflammatory effects.

The question will be where can we use these findings and can we diminish the bed proinflammatory effects of high animal protein meals with antioxidant-rich food. In other words, can we still eat meat but also add some high antioxidant vegetables or fruits in the same meal to avoid the risk (8).

There is no RDA for the antioxidant intake and it is a more complex topic. If you want to learn more about it you can read in Part 3 of the book series (Go Vegan? Review of Science Part 3, By Milos Pokimica).

There was a large number of studies done on a topic and the conclusion is yes, we can, but only to some extent. Avoidance of toxin exposure is our primary goal. If you really have to eat animal products then at least incorporate an adequate amount of anti-inflammatory food sources and calculate your optimal ORAC (Oxygen radical absorbance capacity) intake. This would not completely negate the toxicity of dead meat bacteria endotoxemia exposure. These toxins are very potent and hard to detoxify. There are genetically susceptible individuals that have a harder time detoxifying these compounds. Antioxidant-rich food will lower the risk to a relevant degree and my advice is to optimize the antioxidant intake.

The best course of action would be to have a whole food plant-based diet that has an optimal level of ORAC units and a wide range of proinflammatory food sources with an adequate level of all essential micronutrients.

This is what the British Journal of Nutrition has to say about it.

“Postprandial (fed) state is a pro-oxidant state. The postprandial period is a time of active oxidative metabolism and formation of ROS (free radicals). There is increasing evidence that the postprandial state is an important contributing factor to chronic disease. Two main questions are posed: first, what is the role of plant foods, specifically fruits rich in complex and simple phenolic compounds in postprandial metabolic management; and second, does the evidence support consuming these fruits with meals as a practical strategy to preserve health and lower risk for disease? The collected data suggest that consuming phenolic-rich fruits increases the antioxidant capacity of the blood, and when they are consumed with high fat and carbohydrate ‘pro-oxidant and pro-inflammatory’ meals, they may counterbalance their negative effects. Given the content and availability of fat and carbohydrate in the Western diet, regular consumption of phenolic-rich foods, particularly in conjunction with meals, appears to be a prudent strategy to maintain oxidative balance and health.“

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Sources:

Passages selected from a book: “Go Vegan? Review of Science: Part 1” [Milos Pokimica]

  1. Endotoxin-induced Organ Injury doi: 10.1097/00003246-199302001-00005
  2. Potential Role of Endotoxin as a Proinflammatory Mediator of Atherosclerosis doi: 10.1161/01.ATV.0000147534.69062.dc
  3. High fat intake leads to acute postprandial exposure to circulating endotoxin in type 2 diabetic subjects doi: 10.2337/dc11-1593
  4. The Capacity of Foodstuffs to Induce Innate Immune Activation of Human Monocytes in Vitro Is Dependent on Food Content of Stimulants of Toll-like Receptors 2 and 4 doi: 10.1017/S0007114510003004
  5. A High-Fat Meal Induces Low-Grade Endotoxemia: Evidence of a Novel Mechanism of Postprandial Inflammation doi: 10.1093/ajcn/86.5.1286.
  6. High-fat Meal Induced Postprandial Inflammation doi: 10.1002/mnfr.201300104.
  7. Dietary Cocoa Reduces Metabolic Endotoxemia and Adipose Tissue Inflammation in High-Fat Fed Mice doi: 10.1016/j.jnutbio.2013.12.004.
  8. Postprandial metabolic events and fruit-derived phenolics: a review of the science. doi: 10.1017/S0007114510003909

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Are we omnivores, carnivores or herbivores? It’s important for animals to eat what they are physiologically and anatomically designed to eat, to improve the chances of survival and health. So, what are humans designed to eat? Dr. Sofia Pineda Ochoa discusses this often misunderstood topic.

The first part of the transcript is included below for reference, and the full transcript (which is too long to have here) is available on our website, along with sources and credits, at this link: http://meatyourfuture.com/2015/09/herbivores-carnivores/

[The following transcript is an approximation of the audio in  video. To hear the audio and see the accompanying visuals, please play the video.]

PARTIAL TRANSCRIPT:

Are humans herbivores, carnivores or omnivores? It’s very important for a given animal to eat what they are physiologically and anatomically designed to eat, to improve the chances of survival and health. So, what are humans designed to eat?

When looking at a species to determine what they are in terms of carnivore, omnivore or herbivore, we can look at their behavior or we can look at their biology. From a behavioral standpoint, humans behave as omnivores because we observe many humans in their behavior eating a wide variety of both animal and plant-based foods. Biologically, however, from a physiologic and anatomic standpoint, it’s a different story.

Dr. Williams C. Roberts from the National Institutes of Health and Baylor University — who is the editor-in-chief of the American Journal of Cardiology and one of the most prominent cardiologists in the world with over 1,500 publications in peer reviewed medical journals — summarized our answer very nicely. He wrote:

“Although most of us conduct our lives as omnivores, in that we eat flesh as well as vegetables and fruits, human beings have characteristics of herbivores, not carnivores. The appendages of carnivores are claws; those of herbivores are hands or hooves. The teeth of carnivores are sharp; those of herbivores are mainly flat (for grinding). The intestinal tract of carnivores is short (3 times body length); that of herbivores, long (12 times body length). Body cooling of carnivores is done by panting; herbivores, by sweating. Carnivores drink fluids by lapping; herbivores, by sipping. Carnivores produce their own vitamin C, whereas herbivores obtain it from their diet. Thus, humans have characteristics of herbivores, not carnivores.”

That’s right. Humans have characteristics of herbivores, not carnivores or omnivores — because omnivores, like bears and raccoons, actually retain most of the carnivorous characteristics, so that they are still able to digest and hunt their prey, and do so effectively.

[Remainder of transcript, along with sources and credits, available here: http://meatyourfuture.com/2015/09/herbivores-carnivores]

Are we omnivores, carnivores or herbivores? It’s important for animals to eat what they are physiologically and anatomically designed to eat, to improve the chances of survival and health. So, what are humans designed to eat? Dr. Sofia Pineda Ochoa discusses this often misunderstood topic.

The first part of the transcript is included below for reference, and the full transcript (which is too long to have here) is available on our website, along with sources and credits, at this link: http://meatyourfuture.com/2015/09/herbivores-carnivores/

[The following transcript is an approximation of the audio in video. To hear the audio and see the accompanying visuals, please play the video.]

PARTIAL TRANSCRIPT:

Are humans herbivores, carnivores or omnivores? It’s very important for a given animal to eat what they are physiologically and anatomically designed to eat, to improve the chances of survival and health. So, what are humans designed to eat?

When looking at a species to determine what they are in terms of carnivore, omnivore or herbivore, we can look at their behavior or we can look at their biology. From a behavioral standpoint, humans behave as omnivores because we observe many humans in their behavior eating a wide variety of both animal and plant-based foods. Biologically, however, from a physiologic and anatomic standpoint, it’s a different story.

Dr. Williams C. Roberts from the National Institutes of Health and Baylor University — who is the editor-in-chief of the American Journal of Cardiology and one of the most prominent cardiologists in the world with over 1,500 publications in peer reviewed medical journals — summarized our answer very nicely. He wrote:

“Although most of us conduct our lives as omnivores, in that we eat flesh as well as vegetables and fruits, human beings have characteristics of herbivores, not carnivores. The appendages of carnivores are claws; those of herbivores are hands or hooves. The teeth of carnivores are sharp; those of herbivores are mainly flat (for grinding). The intestinal tract of carnivores is short (3 times body length); that of herbivores, long (12 times body length). Body cooling of carnivores is done by panting; herbivores, by sweating. Carnivores drink fluids by lapping; herbivores, by sipping. Carnivores produce their own vitamin C, whereas herbivores obtain it from their diet. Thus, humans have characteristics of herbivores, not carnivores.”

That’s right. Humans have characteristics of herbivores, not carnivores or omnivores — because omnivores, like bears and raccoons, actually retain most of the carnivorous characteristics, so that they are still able to digest and hunt their prey, and do so effectively.

[Remainder of transcript, along with sources and credits, available here: http://meatyourfuture.com/2015/09/herbivores-carnivores]

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YouTube Video UExXSlpBTjg5dURrWGF0Wkl0aDlDZG55UE1ZcEZ5dUlrbC5DRUQwODMxQzUyRTlGRkY3

Are humans omnivores, carnivores or herbivores?

GoVeganWay 351K views September 4, 2021 12:32 am

How much is a human life worth? An innovative cancer therapy promises to save lives. But it is extremely expensive. Will the insurance companies pay for it? What is the manufacturer's return on investment? And do lobbyists drive up prices?

In 2018, the Kymriah gene therapy was approved in Europe. Immune cells are taken from the patient, genetically reprogrammed into cancer killer cells and returned to the patient as an infusion. The results of the Kymriah study only cover a period of 18 months. In 40 percent of patients, lymph gland cancer does not return during this time. It is not clear whether Kymriah has a long-term effect. The Swiss pharmaceutical company Novartis offers the new therapy - it costs 370,000 Swiss francs per patient. Health insurance companies are not usually prepared to pay that much and are complaining about a lack of transparency. 

But the killer cells were not invented in the Novartis laboratories, but at a US university. When Professor Carl June started his research almost 30 years ago, no pharmaceutical company was interested. It was only thanks to funding from tax money and donations that he was able to develop Kymriah at all. After a story went around the world about a girl with leukemia whose cancer disappeared thanks to Kymriah, the pharmaceutical company contacted Novartis and secured exclusive marketing rights. To launch Kymriah on the market, Novartis funded the necessary clinical trials. It's not an isolated incident: Over 60% of newly approved medicines in the US are developed by small biotech companies or universities. Pharmaceutical companies today frequently act as capital providers, cooperating with universities or buying up biotech companies. 

A paradigm shift has taken place in the pharmaceutical industry: Whereas high drug prices used to be justified by research costs, the industry is now using the value of gained lifetime to argue its case. 

 -------------------------------------------------------------------

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How much is a human life worth? An innovative cancer therapy promises to save lives. But it is extremely expensive. Will the insurance companies pay for it? What is the manufacturer's return on investment? And do lobbyists drive up prices?

In 2018, the Kymriah gene therapy was approved in Europe. Immune cells are taken from the patient, genetically reprogrammed into cancer killer cells and returned to the patient as an infusion. The results of the Kymriah study only cover a period of 18 months. In 40 percent of patients, lymph gland cancer does not return during this time. It is not clear whether Kymriah has a long-term effect. The Swiss pharmaceutical company Novartis offers the new therapy - it costs 370,000 Swiss francs per patient. Health insurance companies are not usually prepared to pay that much and are complaining about a lack of transparency.

But the killer cells were not invented in the Novartis laboratories, but at a US university. When Professor Carl June started his research almost 30 years ago, no pharmaceutical company was interested. It was only thanks to funding from tax money and donations that he was able to develop Kymriah at all. After a story went around the world about a girl with leukemia whose cancer disappeared thanks to Kymriah, the pharmaceutical company contacted Novartis and secured exclusive marketing rights. To launch Kymriah on the market, Novartis funded the necessary clinical trials. It's not an isolated incident: Over 60% of newly approved medicines in the US are developed by small biotech companies or universities. Pharmaceutical companies today frequently act as capital providers, cooperating with universities or buying up biotech companies.

A paradigm shift has taken place in the pharmaceutical industry: Whereas high drug prices used to be justified by research costs, the industry is now using the value of gained lifetime to argue its case.

-------------------------------------------------------------------

DW Documentary gives you knowledge beyond the headlines. Watch high-class documentaries from German broadcasters and international production companies. Meet intriguing people, travel to distant lands, get a look behind the complexities of daily life and build a deeper understanding of current affairs and global events. Subscribe and explore the world around you with DW Documentary.

Subscribe to:
DW Documentary: https://www.youtube.com/channel/UCW39zufHfsuGgpLviKh297Q?sub_confirmation=1#

DW Documental (Spanish): https://www.youtube.com/dwdocumental
DW Documentary وثائقية دي دبليو: (Arabic): https://www.youtube.com/dwdocarabia

For more visit:
http://www.dw.com/en/tv/docfilm/s-3610
Instagram:
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Facebook:
https://www.facebook.com/dw.stories

DW netiquette policy: https://p.dw.com/p/MF1G

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The power of the pharmaceutical companies | DW Documentary

GoVeganWay 648.6K views September 4, 2021 12:24 am

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